Correct Premature Ejaculation in Thailand

Premature Ejaculation Surgery is a surgical treatment used to treat premature ejaculation (PE) problem.

Premature ejaculation (PE) is a prevalent male sexual problem. Premature ejaculation (PE) is one of the most frequent male sexual problems, estimated to affect 4-39 percent of men in the general population.

Ejaculation is a reflex that consists of sensory receptors and areas, afferent pathways, cerebral sensory areas, cerebral motor centers, spinal motor centers, and efferent paths. Normal antegrade ejaculation consists of three essential mechanisms: emission, ejection, and orgasm. Emission is the outcome of a sympathetic spinal cord response triggered by genital and/or cerebral erotic impulses, which entails the successive contraction of accessory sexual organs. Initial voluntary emission control is significant, but it gradually reduces until ejaculatory inevitability. Ejection also triggers a sympathetic spinal cord reaction over which there is little or no deliberate control. Ejection is characterized by bladder neck closure, rhythmic contractions of the bulbocavernous, bulbospongiosus, and other pelvic floor muscles, as well as relaxation of the external urinary sphincter.Orgasm is caused by brain processing of pudendal nerve sensory inputs resulting from increased pressure in the posterior urethra, sensory stimuli from the veramontanum, and contraction of the urethral bulb and sexual organs.

The ejaculatory reflex is primarily regulated by a complex interplay of central serotonergic and dopaminergic neurons, with additional participation from cholinergic, adrenergic, nitrergic, oxytocinergic, galanergic, and GABAergic neurons. The brain events that occur during ejaculation, as well as the abnormalities found in males with PE, have not been fully described using positron emission tomography (PET) or functional magnetic resonance imaging (fMRI). Several forebrain regions, including as the medial preoptic area (MPOA) and the nucleus paragigantocellularis, integrate seminal emission and ejection into the intricate pattern of copulatory behavior. The descending serotonergic pathways from the nPGI to the lumbosacral motor nuclei tonically inhibit ejaculation. The MPOA inhibits the nPGI, which enhances ejaculation. In male rats, a population of lumbar spinothalamic neurons has been found (LSt cells), which play an important role in ejaculation generation. LSt cells transmit projections to autonomic nuclei and motoneurons involved in the emission and expulsion phases, as well as receiving sensory projections from the pelvis.Ascending fibres from the spinal cord engage several brain areas following ejaculation, which may play a role in satiety and postejaculatory refractory period.

During this procedure:

The surgery was conducted under local anesthetic with 2% Lidocaine. The surgeon will mark the incision. A transverse incision of 2 cm was made 1.5 cm proximal to the coronal sulcus, followed by a sharp dissection under the Buck fascia. To create the inner condom, transfer a 4 × 6 cm sheet of type J-1 ADM to the subcutaneous pocket and secure with vicryl sutures. The penis is immobilized with an elastic bandage for 7 days, and the sutures are then removed after 10 days.

Recovery Period:

The average follow-up period was 6 months (range: 2–12 months). Penile edema was noticed in the first 2 to 4 weeks after surgery. Except for that, none of the guys experienced any other issues such as aberrant incision healing, implant exposure, infection, hematoma, or seroma.